Download A4Medicine Mobile App
Empower Your RCGP AKT Journey: Master the MCQs with Us! 🚀
Body‑hair changes are an important clinical clue to metabolic and endocrine disorders.
Included in the RCGP curriculum under Metabolic problems and endocrinology.
Presentations range from:
Excess hair growth – hirsutism (male-pattern) or hypertrichosis (generalised).
Hair loss or thinning – alopecia (patchy or diffuse).
May indicate underlying hormonal imbalance (e.g., androgen excess, thyroid dysfunction, adrenal or pituitary disorders).
Can be physiological (puberty, pregnancy, menopause) or pathological (PCOS, Cushing’s, androgen-secreting tumours).
Often associated with psychological distress → impacts quality of life.
Early recognition → appropriate investigation, management, and referral.
Anagen (growth phase): Active hair production; lasts years on scalp.
Catagen (involution phase): Follicle regression; lasts weeks.
Telogen (resting phase): Shedding phase; lasts months.
Convert vellus hair → terminal hair (thicker, pigmented).
↑ Sebum production and anagen duration on body hair.
Shorten anagen on scalp → may contribute to androgenic alopecia.
Follicle sensitivity varies by individual and ethnicity.
Ferriman–Gallwey score: Rates 9 androgen‑sensitive areas (e.g., upper lip, chin, chest).
Score ≥8 = hirsutism in UK women (NICE CKS).
Thyroid hormones: Regulate follicle metabolism and hair cycle.
Cortisol, GH, prolactin: Direct impact...
Try our Free Plan to get the full article.